This summer, a US based bio-technology company will proceed to test the effectiveness of human embryonic stem cell therapy on patients with spinal cord injuries. The much anticipated research project will be the world's first study of treatments which incorporate human stem cells to treat spinal cord injuries.
Researchers at MIT have discovered that Sirtuin1, a protein encoded by the SIRT1 gene, promotes synaptic plasticity and boosts memory.
NT-020, a combination of nutrients, increased neural stem cell proliferation, neurogenesis and helped improve memory in the aging brains of mice.
Genetics research into the emotional responses of bullying shows that many bullied victims that later experience more emotional problems have genetic similarities.
A large risk genetic marker of schizophrenia has been discovered by scientists. The researchers found that a specific deletion at 3q29, which contains two genes already known to be associated with intellectual disabilities , PAK2 and DLG1, increased the odds of schizophrenia symptoms by nearly 17 times those with no deletion.
New research points to a DNA sequence that causes the DUX4 gene to become more active in producing proteins that are toxic to muscle cells, leading to a form of muscular dystrophy.
New research proposes that academic performance of adolescents is linked to specific dopamine gene variations. Researchers also suggest a correlation exists between the variants of dopaminergic genes a student possesses and performance in specific subject areas.
Neuralstem has released a new report detailing positive results for stem cell treatments. Initial findings show that not only do implanted human spinal cord-derived stem cells survive, but also differentiate into neurons in rats brains affected by strokes. This finding could potentially provide new therapies for treating strokes.
Researchers have discovered that disabling the RGS14 gene in mice can make them smarter. When the RGS14 gene was disabled within the CA2 region of the hippocampus, researchers found that mice were better able to remember objects they had explored and learn to navigate mazes better than regular mice.
Neuroscientists at USC have discovered that carbonated drinks set off the same pain sensors in the nasal cavity as mustard, albeit at a lower intensity. During experimentation, researchers flowed carbonated saline over a dish containing nerve cells taken from the sensory circuits in the nose and mouth. They discovered that the gas activated a specific cell which serve as general pain sensors and expresses the TRPA1 gene.