A study of the salamander brain has led researchers at Karolinska Institutet to discover a hitherto unknown function of the...
As Parkinson’s Awareness Month gets underway, a Canadian-led international study is providing important new insight into Parkinson’s disease and paving...
Communication errors between brain cells may lead to neurodegenerative diseases such as Parkinson’s Patrik Verstreken (VIB/K.U.Leuven) has discovered the mechanism...
Oxford University scientists have developed a new method for delivering complex drugs directly to the brain, a necessary step for...
New Parkinson's disease research suggests defective regulation of microtubules may be responsible for some cases of Parkinson's disease.
Scientists show that SR-3306 delivered orally to mice and rat models of Parkinson's disease was able to protect brain cells from neurodegeneration. SR-3306 inhibits the c-jun-N-terminal kinases (JNK) class of enzymes, previously shown to play large roles in neuron survival.
Scientists have shown that stem cells delivered to rats via a nasal spray lead to an improvement of motor functions...
Parkinson’s disease may be caused by oxidative stress within cells due to defective nucleoli. Dopamine producing neurons are particularly sensitive...
Scientists discovered the reaction which starts the formation of amyloid fibers. Amyloid fibers are problematic in many diseases and disorders...
Max Planck researchers discovered that large, less hydrophobic proteins with a significant increase of disorder in their structures were more...
Huntington’s disease research using simple organisms such as baker’s yeast has yielded new information about underlying mechanisms of Huntington’s disease...
Neuroscience researchers have identified a new molecular pathway underlying Parkinson's disease. The pathway involves polyamines, which were discovered to be responsible for increased build-up of other toxic proteins in neurons. The research also suggests polyamine lowering drugs could have a protective effect from Parkinson's disease.
