The biological clock doesn't stop working for those with Alzheimer's disease, but it does detach from sleep wake cycles, a new report claims.
Researchers have discovered a potential drug which can force BACE into different parts of a cell, reducing its ability to produce amyloid beta.
Researchers transform iPSCs into neurons to test the efficacy of drugs to treat Alzheimer's disease.
Researchers discover a way to traverse the blood-brain barrier and retinal-blood barrier to deliver a molecule which degrades the main component of Alzheimer's plaques.
Researchers discover by looking at amyloid-beta and tau independently that the interaction between the proteins could be the key to the cognitive decline associated with Alzheimer's disease. As the interaction between the two proteins increased, the progression of Alzheimer's worsened.
Researchers have developed an experimental drug, NitroMemantine, to boost brain synapses lost in Alzheimer's disease. The drug stops the destructive cascade of brain changes which destroy connections between neurons that leads to memory loss and cognitive decline.
Using transgenic mice with specially designed gene fragments derived from bacteria and human cells, researchers were able to control the production of amyloid beta peptide using antibiotics.
Researchers discovered a way to stimulate the brain's natural defense mechanisms in mice with Alzheimer's disease symptoms. This major breakthrough opens the door to the development of a treatment for Alzheimer's disease and a vaccine to prevent the illness.