Combining AI and robotics technology, researchers have identified new cellular characteristics of Parkinson's disease in skin cell samples from patients.
Older adults who take statin drugs have a lower risk of developing Parkinson's disease or parkinsonism symptoms later in life compared to those who do not take statins. Researchers speculate this may be because statins have a neuroprotective effect on arteries in the brain.
People with Parkinson's disease have a clear genetic signature of the disease in their memory T-cells.
The presence of p-syn on the skin distinguishes people with Parkinson's disease from those with Parkinson's symptoms associated with other neurological disorders.
People who suffer from a neurological or mental health condition are at increased risk of developing another disorder later in life. Parkinson's disease patients are four times more likely to develop dementia, and those with mental health disorders were also at greater risk of developing dementia later in life.
Hearing loss and epilepsy are early features of Parkinson's disease, a new study reveals.
Kushen, a dried root of a pea plant, may alleviate symptoms of neuroinflammation in Parkinson's disease when combined with an enzyme inhibitor.
Carnosic acid, a compound found in the herb rosemary, can block the interaction with the COVID-19 SARS_CoV_2 spike protein and ACE2, the protein receptor the virus uses to gain access into cells. The compound appears to have anti-inflammatory effects and has previously been associated with a reduction in Alzheimer's symptoms.
Parkinson's patients who increase the number of flavonoid-rich foods they consume as part of their diet have a lower mortality risk than those who don't.
New findings reveal exercise increases levels of endocannabinoids in the body. The findings may explain some of the beneficial effects of exercise on the brain and body.
Findings reveal a common Parkinson's disease genetic mutation drives mislocalization of iron in activated microglia.
The COVID causing SARS-CoV-2 protein interacts with alpha-synuclein, speeding up the formation of amyloid plaques, a new study reports.
