Researchers pinpoint the role of the Arl13b gene in the formation and proper placement of neurons during brain development.
Researchers discover Sturge-Weber syndrome, a neurological and skin disorder, is caused by a genetic mutation which occurs prior to birth.
A new study finds children with autism see simple movements twice as quickly as their non-autistic peers. This hypersensitivity to motion could provide clues to one of the fundamental causes of autism.
New research suggests children with ASD spend more time playing video games and watching TV than participating in social media and pre-social activities compared to their typically developing peers.
A new study reveals 70 percent of children diagnosed with autism spectrum disorders, and who have a history of severe language delay, achieved fluent speech by the age of eight.
Researchers find that, compared with neurotypical children, those with autism have multiple redundant connections between neighboring brain areas at the expense of long-distance links.
New research suggests environmental exposure to bisphenol A (BPA), a chemical commonly found in plastics and resins, could suppress a gene critical to nerve cell function and the development of the central nervous system. Exposure to BPA could predispose humans to a number of neurodevelopmental and other health disorders.
Genes linked to autism and schizophrenia are only switched on during the early stages of brain development, according to a study in mice led by researchers at the University of Oxford.
Future research into the underlying causes of neurological disorders such as autism, epilepsy and schizophrenia, should greatly benefit from a first-of-its-kind atlas of gene-enhancers in the cerebrum (telencephalon). This new atlas identifies and locates thousands of gene-regulating elements in a region of the brain that is of critical importance for cognition, motor functions and emotion.
A study shows another family of proteins linked to neurodevelopmental disorders regulates the function of neuroligins and neurexins in order to suppress the development of inhibitory synapses.
A new Caltech study suggests that specific changes in an overactive immune system can indeed contribute to autism-like behaviors in mice, and that in some cases, this activation can be related to what a developing fetus experiences in the womb.