Observing whole-brain activity in live zebrafish, researchers have discovered how information stored as long-term memory in the cerebral cortex is processed to guide behavioral choices.
Researchers have identified biomarkers for Alzheimer's disease which accurately predict the development of the disease years prior to symptoms developing.
A drug developed by scientists at the Salk Institute for Biological Studies, known as J147, reverses memory deficits and slows Alzheimer's disease in aged mice following short-term treatment.
Researchers question why numerous different RNAs are transported to synapses. One reason, they suggest, is that they are stored to later help maintain long-term memory.
Researchers describe in detail the underlying neurobiology of the "world's second most famous amnesiac", EP, who suffered profound memory loss after damage to key portions of his brain.
Using the nerve cells of sea snails, researchers reverse memory loss by determining when cells were primed for learning. Retraining cells with the use of an optimized training schedule helped compensate for memory loss.
Adapting two dimensional culture methods to grow 3-D neuron structures from induced pluripotent stem cells, researchers may be able to model and study neurodegenerative disorders such as Alzheimer's disease.
A new study identifies a potential treatment window of several years for plaques in the brain considered to cause memory loss in diseases such as Alzheimer's.
A new study offers evidence of source memory in nonhuman animals.
Researchers have found compelling evidence that older adults can eliminate forgetfulness and perform as well as younger adults on memory tests. The findings have intriguing implications for designing learning strategies for older adults.
Researchers combined genetic data with neuroimaging in order to identify genes associated with amyloid plaque deposits in Alzheimer's disease patients. The study is believed to be the first genome-wide association study of plaque deposits using a specialized PET scan tracer that binds to amyloid.
A new study suggest that along with amyloid deposits, white matter hyperintensities may be a second necessary factor for the development of Alzheimer’s disease.
