Axitinib, a chemotherapy drug that inhibits the growth of new blood vessels in the brain, improved memory and cognition in mouse models of Alzheimer's disease.
Fenchol, a natural compound found in basil and other plans, appears to have a neuroprotective effect against Alzheimer's disease. Fenchol reduces the formation of zombie neural cells and increases the degradation of non-functional amyloid beta, allowing for quicker clearance of the protein from the brain.
Study shows chronic hyperglycemia impairs working memory performance and alters fundamental aspects of working memory brain networks. The findings strengthen the link between type 2 diabetes and Alzheimer's disease.
PET study finds early accumulation of the tau protein in the brain is a better predictor of Alzheimer's associated memory decline than amyloid plaque accumulation or cerebrospinal fluid biomarkers.
Newly designed antibodies that bind to small clumps of amyloid-beta may keep the progression of Alzheimer's disease in check.
Levetiracetam, an inexpensive drug commonly used in the treatment of epilepsy, improved memory, learning, and cognition for Alzheimer's patients who experience epilepsy activity in their brains.
Depression experienced during early adulthood increases the risk of developing cognitive impairment and dementia later in life, a new study reports.
Feeling content in life slows cognitive decline in older adults, a new study reports. Researchers say the odds of older adults developing cognitive impairment and dementia were reduced in those who reported better psychological well-being.
A new blood test that uses atomic force microscopy technology appears to be effective as an Alzheimer's diagnostic test.
A newly developed two-minute EEG test effectively picks up on early memory impairments associated with Alzheimer's disease.
Greater radiodensity of perivascular adipose tissue in women during midlife was associated with decreased working memory performance later in life.
Researchers identified specific receptors for acetylcholine that reroute information flow through memory circuits in the hippocampus. The findings could have implications for the development of drugs to help enhance or protect memory from diseases associated with cognitive decline.
