Researchers found an association between low vitamin D levels and reduced brain volume. Lower vitamin D was also linked to an increased risk of stroke and dementia. Up to 17% of dementia cases could be prevented by increasing vitamin D.
Adopting a healthy lifestyle or adopting minor lifestyle changes helps reduce the risks of developing Alzheimer's disease, especially for those from a lower socioeconomic background.
The prevalence of brain changes associated with LATE, a form of dementia first identified in 2019, may be as high as 40% in older adults and 50% in those with Alzheimer's disease.
People who suffer cardiometabolic disorders, such as stroke, diabetes, or a heart attack, either as stand-alone conditions or a combination of the conditions, have an increased risk of developing dementia regardless of whether or not they have a genetic predisposition for neurodegeneration.
Despite speculation that inflammation associated with the shingles virus can increase dementia risk, researchers found little evidence to substantiate the claim.
People with higher levels of omega-3 DHA in their blood are 49% less likely to develop dementia than those with lower levels. Researchers say adding additional omega-3 DHA to the diet, especially in those with the Alzheimer's associated Apoe4 gene, could slow the development and progress of dementia.
Rapamycin, a drug approved for the treatment of cancer and transplant patients increases amyloid-beta protein plaques in the brains of mice.
The heat produced by amyloid-beta aggregation may cause other, healthy amyloid-beta to aggregate, causing more and more aggregates to form. However, with the addition of a novel drug compound, amyloid-beta aggregation can be stopped and the cell temperature lowered.
White matter may be the key to understanding dementia, TBI, and numerous other neurological disorders.
Fragmented tau that accumulates in neurons in those with Alzheimer's disease may be a new target for drugs to treat the neurodegenerative disease.
A new mouse study reveals a breakdown in the process that clears brain cells of waste products precedes the buildup of amyloid plaques associated with Alzheimer's disease.
Study evaluates whether problems in facial processing and recognition in Alzheimer's disease are a result of memory impairment or visual processing deficits.
