Researchers have identified a circuit within the brain that may be responsible for respiratory dysfunction and sudden death associated with Dravet syndrome.
Researchers say serotonin, a chemical commonly associated with mood regulation, may play in breathing problems associated with epileptic seizures. Those who had higher levels of serotonin following a seizure than before the event were less likely to experience interrupted breathing.
Mice expressing a specific variant of the Scn1a gene associated with Dravet syndrome exhibited spontaneous seizures, disordered breathing and died prematurely.
An opinion article reports on a potential common cause for both SIDS and SUDEP. The reason sudden death may occur is due to a person's inability to wake when CO2 levels in the blood rise, as a result of dysfunctional serotonin neurons in the midbrain.
A new study reports a mutation in a gene that helps regulate calcium in neurons can trigger blackouts in the brain-stem and increase the rick of sudden, unexpected death.
According to a new study, the heart rate during sleep in children with epilepsy is significantly slower than in those without the condition.
For the first time, researchers have successfully stimulated a human amygdala and recorded breathing loss. Their research provides important insight into SUDEP.
By turning skin cells of patients with Dravet syndrome into neurons, researchers create a miniature testing ground for epilepsy
