A new technology known as sonogenetics can control neural activity by using sound frequencies. The technology could be used to non-invasively treat a range of neurological conditions, including Parkinson's disease and epilepsy.
A soft neural implant that can be controlled by a smartphone is capable of drug delivery and optogenetics. The technology could speed up efforts to uncover the causes of neurological and psychological disorders.
Using optogenetics, researchers stimulated neurons in the visual cortex of mice to induce illusory images in the animals' minds.
Using holographic optogenetics, researchers were able to control visual behavior in mice by activating neurons in the visual cortex.
Serotonin produced by the raphe is critical for sleep in both mice and zebrafish. The firing of neurons in the raphe and the release of serotonin may help the brain build up better sleep pressure. The results may explain why some sleep-related side effects of antidepressants increase serotonin in the brain.
Artificially prolonging hippocampal sharp wave ripples improves working memory.
Using optogenetics to stimulate different areas of the hippocampus has the ability to enhance or suppress memories in mice. The findings could have implications for suppressing memories associated with traumatic events in PTSD, and also in enhancing cognitive ability or improving memory for those with neurodegenerative diseases, in the future.
Using optogenetics to 'switch off' the insular cortex in mice experiencing a painful event resulted in a less fearful reaction to pain. The ability of the mice to learn avoidance from the pain event was also reduced.
SatB2PBN-expressing neurons in the parabrachial nucleus play a key role in processing and encoding sweet tastes. The SatB2PBN neurons relay sweet taste signals from the gustatory thalamus to the cortex in mouse models.
Study reveals anesthesia activated neurons have been discovered in the hypothalamus. Activation of AANs promotes slow-wave sleep, extending the effects of anesthesia, while inhibition of AANs shortens the duration of general anesthetics and disrupts natural sleep.