Mice exposed to dust from the WTC showed impairments in both long and short-term memory, and spatial recognition, as well as alterations to genes associated with immune-inflammatory response and blood-brain barrier dysfunction. Findings suggest first responders who were exposed to dust from the WTC experience a peripheral-brain immune inflammatory response that leads to cognitive decline.
People with subretinal drusenoid deposits (SDD), a form of age-related macular degeneration, are more likely to have underlying heart damage as a result of heart failure or heart attacks, or other forms of cardiovascular disease associated with increased stroke risk.
Neurons derived from stem cells of combat veterans with PTSD react differently to stress hormones than those derived from veterans without PTSD. The findings shed new light on how genetics can cause some to develop PTSD following a traumatic event.
Sleep deprivation alters the structure of DNA inside immune cells and increases the number of immune cells, which causes them to overreact and spark inflammation. The study found catching up on sleep does not reverse this effect.
Researchers have identified a neural mechanism that supports advanced cognitive functions such as planning and problem-solving. The mechanism distributes information from a single neuron to larger neural populations in the prefrontal cortex.