Study identifies a group of proteins in the cerebrospinal fluid that could serve as biomarkers for inflammation in the brain.
Neuroimaging technology allowed researchers to capture the activity of microglia and astrocytes in the brain. The researchers were able to quantify alterations in the morphology of different cell populations implicated in neuroinflammation.
Glycan, a special sugar protein, appears to play a significant role in the development of Alzheimer's disease.
Researchers found idiopathic autism occurs as the result of epigenetic abnormalities in hematopoietic cells during fetal development, leading to immune dysregulation in the brain and gut.
New findings reveal phagocytes do not fully mature until after birth, contradicting previous assumptions that they mature during embryonic development.
An abundance of newly acquired mutations in the mutations that occur at an accelerated speed is a telling pattern of Alzheimer's disease, researchers report.
Researchers report the intrinsic excitability of neurons in response to immune cell reaction to bacteria depends on the different neuron subtypes in rats.
Tau-tangles trigger the inflammatory activation of microglia via the NF-κB pathway. Inhibiting the microglia NF-κB signaling pulled the immune cells out of their inflammatory state and reversed learning and memory problems in tau-based Alzheimer's mouse models.
Researchers identified a link between major depressive disorder and mechanical alterations in blood cells.
Microglia dysfunction in those who experienced early life adversity prompts an aberrant stress response during adulthood that may be linked to mental illness.
A molecular process caused by the TREM2 gene mutation in the brain's microglia increases the risk of Alzheimer's disease, a new study reports.
Niaspan, an FDA-approved drug, limits disease progression in lab models of Alzheimer's disease.
