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Featured Neurology Neuroscience Open Neuroscience Articles

·October 19, 2021·4 min read

Viral Infections Could Promote Neurodegeneration

Some viral infections could increase intercellular spreading of protein aggregates associated with neurodegenerative disorders, increasing the risk for developing Alzheimer's, Parkinson's, and other neurodegenerative diseases.

This shows dying neurons and areas of brain tissue infected with SARS_CoV_2

Featured Neurology Neuroscience

·January 12, 2021·3 min read

SARS_CoV_2 Can Infect Neurons and Damage Brain Tissue

SARS_CoV_2, the virus responsible for COVID-19, can directly enter the nervous system. The virus can infect the brain, causing alterations in blood vessels and directly disrupt oxygen supply to the organ.

Featured Neurology Neuroscience

·January 5, 2021·5 min read

How COVID-19 Is Likely to Impact the Brain

Using over a century of data from other pandemics, and applying knowledge about the current COVID-19 infection, researchers predict the long term effects coronavirus will have on the brain and nervous system.

Featured Genetics Neuroscience Open Neuroscience Articles

·August 25, 2020·3 min read

Scientists Prove SARS-CoV-2 Potential to Infect Human Brain Organoids

SARS-CoV-2, the virus responsible for COVID-19, can infect human neural progenitor cells and brain organoids. The findings back previous research, finding coronavirus can infect the human brain.

This is a drawing of a man in a facemask

Featured Genetics Neuroscience

·July 24, 2020·7 min read

How COVID-19 Causes Smell Loss

Infection of non-neuronal supporting cells in the nose and forebrain may be responsible for the olfactory problems associated with COVID-19. Findings suggest olfactory sensory neurons are not vulnerable to coronavirus infection as they do not express ACE2.

Featured Neuroscience Open Neuroscience Articles

·June 24, 2020·5 min read

Statin use is linked to lower death rate in hospitalized COVID-19 patients

Cholesterol-lowering statins show promise for the treatment of severe coronavirus infections. Statins lowered the death rate and decreased the need for mechanical ventilation in patients hospitalized for severe COVID-19.

Featured Genetics Neurology Neuroscience

·May 26, 2020·2 min read

Researchers flag similarities between COVID-19 deaths and severe rheumatic illnesses

After natural killer immune cells kill virus-infected cells, T and B immune cells produce cytokines. This makes the immune reaction stronger and results in the cytokine storm associated with severe COVID-19 infection.

Featured Neuroscience Open Neuroscience Articles

·May 20, 2020·3 min read

COVID-19 cytokine storm: Possible mechanism for the deadly respiratory syndrome

SARS-CoV-2, the virus responsible for COVID-19, enters human cells by attaching to ACE2 and utilizing TMPRSS2. Drugs that block ACE2 or inhibit the enzyme could help treat the coronavirus, but only during early infection. As the infection progresses, SARS-CoV-2 becomes engulfed in human cells, reducing the number of ACE2 receptors on a cell and leading to an increase of angiotensin II in the blood. Angiotensin II triggers an inflammatory pathway, providing a positive feedback cycle, named IL-6 amplifier, resulting in excessive immune activation and the cytokine storm associated with severe COVID-19.

Featured Genetics Neuroscience

·May 18, 2020·3 min read

Smokers more likely to express ACE2 protein that coronavirus uses to enter human cells

Smoking increases the gene expression of ACE2, a protein that binds to SARS-CoV-2, increasing the risk of coronavirus infection. Findings suggest long-term smokers could have an increase of ACE2 in the lungs, leading to higher rates of morbidity in COVID-19 patients.

Featured Neurology Neuroscience Psychology

·May 15, 2020·6 min read

The Potential of COVID-19 to Infect the Brain

From losing the sense of taste or smell to developing an increased risk of stroke, researchers investigate both the known and potential long-term implications of COVID-19 infection in the brain.

Featured Neuroscience

·May 12, 2020·2 min read

Exploring why some COVID-19 patients lose their sense of smell

ACE2 and TMPRSS2, two proteins required for SARS-CoV-2 entry, are produced in cells in the nasal cavity that contribute to odor detection. The findings may explain why people with coronavirus often describe the loss of the sense of smell as a symptom of the virus.

Featured Genetics Neuroscience Open Neuroscience Articles

·May 11, 2020·6 min read

Men’s blood contains greater concentrations of enzyme that helps COVID-19 infect cells

Men have higher concentrations of ACE2 in their blood than women. As ACE2 enables coronavirus to infect cells, the findings may explain why men are more susceptible to COVID-19 infection than women.