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Featured Neuroscience Open Neuroscience Articles

·May 20, 2020·3 min read

COVID-19 cytokine storm: Possible mechanism for the deadly respiratory syndrome

SARS-CoV-2, the virus responsible for COVID-19, enters human cells by attaching to ACE2 and utilizing TMPRSS2. Drugs that block ACE2 or inhibit the enzyme could help treat the coronavirus, but only during early infection. As the infection progresses, SARS-CoV-2 becomes engulfed in human cells, reducing the number of ACE2 receptors on a cell and leading to an increase of angiotensin II in the blood. Angiotensin II triggers an inflammatory pathway, providing a positive feedback cycle, named IL-6 amplifier, resulting in excessive immune activation and the cytokine storm associated with severe COVID-19.

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Featured Genetics Neuroscience

·July 24, 2020·7 min read

How COVID-19 Causes Smell Loss

Infection of non-neuronal supporting cells in the nose and forebrain may be responsible for the olfactory problems associated with COVID-19. Findings suggest olfactory sensory neurons are not vulnerable to coronavirus infection as they do not express ACE2.

Featured Neuroscience Open Neuroscience Articles

·May 1, 2020·4 min read

Coronavirus infects cells of the intestine: Stool samples could be used to test for virus

SARS-CoV-2, the virus that causes COVID-19, can infect intestinal cells and multiple in the gastrointestinal system. When researchers added SARS-CoV-2 to intestinal organoids, they noticed rapid infection. Researchers say in addition to nasal and throat swabs, rectal swabs and stool samples could be key for coronavirus testing.

Featured Neuroscience Open Neuroscience Articles

·March 23, 2020·2 min read

ACE inhibitors and angiotensin receptor blockers may increase the risk of severe COVID-19

Researchers confirm ACE inhibitors and ARBS medications may increase the risk of patients with COVID-19 developing more severe symptoms.

Featured Neurology Neuroscience Open Neuroscience Articles

·October 19, 2021·4 min read

Viral Infections Could Promote Neurodegeneration

Some viral infections could increase intercellular spreading of protein aggregates associated with neurodegenerative disorders, increasing the risk for developing Alzheimer's, Parkinson's, and other neurodegenerative diseases.

Featured Neurology Neuroscience Psychology

·May 15, 2020·6 min read

The Potential of COVID-19 to Infect the Brain

From losing the sense of taste or smell to developing an increased risk of stroke, researchers investigate both the known and potential long-term implications of COVID-19 infection in the brain.

Featured Genetics Neuroscience Open Neuroscience Articles

·August 25, 2020·3 min read

Scientists Prove SARS-CoV-2 Potential to Infect Human Brain Organoids

SARS-CoV-2, the virus responsible for COVID-19, can infect human neural progenitor cells and brain organoids. The findings back previous research, finding coronavirus can infect the human brain.

Featured Neuroscience Open Neuroscience Articles

·April 23, 2020·4 min read

Evidence suggests COVID-19 isn’t sexually transmitted

Previous studies report COVID-19 may be present in the testes. Researchers report coronavirus is unlikely to spread via semen, and the chances of the infection being spread through sexual transmission are remote.

Featured Neurology Neuroscience

·January 5, 2021·5 min read

How COVID-19 Is Likely to Impact the Brain

Using over a century of data from other pandemics, and applying knowledge about the current COVID-19 infection, researchers predict the long term effects coronavirus will have on the brain and nervous system.

Featured Neuroscience Open Neuroscience Articles

·June 24, 2020·5 min read

Statin use is linked to lower death rate in hospitalized COVID-19 patients

Cholesterol-lowering statins show promise for the treatment of severe coronavirus infections. Statins lowered the death rate and decreased the need for mechanical ventilation in patients hospitalized for severe COVID-19.

Featured Genetics Neuroscience Open Neuroscience Articles

·May 11, 2020·6 min read

Men’s blood contains greater concentrations of enzyme that helps COVID-19 infect cells

Men have higher concentrations of ACE2 in their blood than women. As ACE2 enables coronavirus to infect cells, the findings may explain why men are more susceptible to COVID-19 infection than women.

Featured Neuroscience Open Neuroscience Articles

·April 29, 2020·2 min read

Tobacco smoking increases lung entry points for COVID-19

Smoking remodels the gene expression of lung cells so that the ACE2 gene is more highly expressed in goblet cells. The effects of smoking on ACE2 pulmonary expression indicates an increase in the overall entry points for coronavirus and increases the risk for viral binding and entry of COVID-19 into the lungs.

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