People with two of the diseases, type 2 diabetes, cardiovascular disease or stroke have double the risk of developing dementia, researchers say.
Only 1 in 3 infants enrolled in the government's WIC program are receiving the daily recommended dose of vitamin d, a new study reports. Vitamin d deficiency plays a role in a number of disorders including rickets, multiple sclerosis, and type 2 diabetes.
Consuming a healthy plant-based diet consisting of fruits, vegetables, nuts, coffee, and legumes lowers the risk of developing type 2 diabetes.
Study shows chronic hyperglycemia impairs working memory performance and alters fundamental aspects of working memory brain networks. The findings strengthen the link between type 2 diabetes and Alzheimer's disease.
Dipeptidyl peptidase-4 inhibitors, a class of drug that reduces blood sugar in type 2 diabetes, were associated with less amyloid accumulation in the brain and slower cognitive decline in patients.
Obese mice treated with the TSLP cytokine showed a significant loss in abdominal fat and weight. The fat loss was not associated with reduced food intake or faster metabolism, instead the cytokine stimulated the immune system to release lipids via the skin's oil-producing sebaceous glands.
Sleep disruptions have been linked to a higher risk of death, especially in those with type 2 diabetes. Researchers found those with type 2 diabetes who reported frequent sleep disruptions, were 87% more likely to die of any cause than those without diabetes or sleep disturbances. Additionally, those with diabetes and sleep disruptions were 12% more die over a nine-year period than those with diabetes alone.
Type 2 diabetes is not only associated with an increased risk of developing Parkinson's disease, but it's also associated with an accelerated progression of Parkinson's symptoms.
Metformin, a drug commonly used to treat diabetes, slows cognitive decline and reduces dementia risk in older people with diabetes.
Older women with type 2 diabetes do not use as much oxygenated blood in their brains as those who do not have the disease. Findings demonstrate alterations in neural blood use are a primary reason for deficits in motor function experienced by those with diabetes.
Researchers demonstrate how a single injection of fibroblast growth factor 1 (FGF1) can restore blood sugar levels to normal for extended periods in rodent models of type 2 diabetes. Studies show how FGF1 affects specific neurons and perineuronal nets to help restore blood sugar levels to normal, thus sending diabetes into remission.