Mouse models of corticospinal injuries reveal adult neurons begin a natural regeneration process by reverting back to an embryonic state. The regeneration is sustained with the help of a gene more commonly associated with Huntington's disease.
Neural progenitor cells generated from skin cell samples of people on the autism spectrum had heightened levels of DNA damage. The damage clustered in 36 of the same genes which had also been damaged in healthy cells exposed to replication stress. Twenty of the genes have previously been linked to the development of autism.
A mutation of the Memo1 gene results in the disruption of radial glial cell scaffold, causing misplaced and disorganized layers. The mutation may be responsible for brain development abnormalities associated with autism.
V2a interneurons could potentially repair spinal cord injuries, researchers report.
Study overturns conventional thought about how axons grow during embryonic development.
Findings could help to identify cells affected by Zika virus, researchers report.
A new study could help explain why children born to mothers infected by Zika are more likely to have microcephaly.
Researchers have successfully transplanted human neurons to treat symptoms of spinal cord injury in mice. The researchers believe this could pave the way for eventual treatment in humans.