Tamapin, an investigational drug derived from Indian red scorpion venom, reverses motor deficits in pre-clinical models of fetal alcohol spectrum disorder.
Study reveals how acetate, a byproduct of alcohol breakdown, travels to the brain's learning system and alters proteins that regulate DNA function.
Teenagers who were exposed to alcohol while in the womb are more likely to have disruptions in neural connections through the corpus callosum, which can lead to cognitive impairment and a range of neurological, and psychological disorders. Researchers recommend pregnant women completely abstain from drinking alcohol.
According to researchers, ibruprofen can reduce neuroinflammation and improve cognitive function in mouse models of FASD.
A new study challenges conventional beliefs about fetal alcohol spectrum disorders. Researchers discovered no differences in microglia activity in the brains of health mice and those exposed to alcohol during early development. The study brings into question the whether the brain's immune system cells are to blame for the neurological damage that occurs as a result of fetal alcohol exposure.
Researchers from Northwestern University reveal that, with the aid of two drugs, it may be possible to repair neurological damage caused as a result of prenatal alcohol exposure. The study reports both drugs help normalize genes that control DNA methyl transferase1 expression, an enzyme critical for brain development.
A new review reports on associations between birth defects in offspring and its father's age, alcohol use and other environmental factors.
A new study shows neurodevelopment is delayed throughout childhood and adolescence for those born with FASD.
Research have identified a signaling pathway that might determine genetic susceptibility for the development of fetal alcohol spectrum disorder (FASD).
