Auditory hallucinations, a common feature of psychosis and schizophrenia, may be the result of increased connectivity between sensory and language processing areas in the brain.
A new study reports hyper-connectivity between substructures of the thalamus, and the cerebral cortex may be responsible for auditory hallucinations associated with schizophrenia.
Studying auditory regions and brain pathways in humans, apes, and monkeys, researchers have identified a language pathway that interconnects the auditory cortex with frontal lobe regions. The language pathway in other primates suggests an evolutionary basis in auditory cognition and vocal communication.
Using Cappella recordings, researchers discover humans have developed complementary neural systems in each hemisphere for auditory stimuli.
When exposed to auditory stimulation, visual areas of the brain decreased in activity. Findings suggest sound can strongly draw attention away from what we are looking at.
Abnormal activation of parvalbumin neurons in the young mouse auditory cortex manifests as a result of maternal neglect.
··4 min read
Combining neuroimaging data with machine learning, researchers report musical pleasure depends on a dynamic interplay between prospective and retrospective states of expectation.
When a person listens to another person talking, their brain waves alter to select specific features of the speaker's voice and tune out other voices.
People with schizophrenia who experience auditory hallucinations have greater activation in specific areas of the auditory cortex in response to sound frequencies. The mapping of sound frequency in the auditory cortex is scrambled in those with schizophrenia, suggesting a disruption in the normal processes for organized sound representation in the brain. As the tonotopic map is established during infancy and remains stable throughout life, the findings suggest vulnerability for auditory hallucinations is linked to defects in the organization of the auditory system during infantile development. This precedes speech development and the onset of psychiatric symptoms.