Extracts from two common wildflowers, tall goldenrod and eagle fern blocked SARS_CoV_2, the virus responsible for COVID-19, from entering human cells. Researchers warn the public they should not consume the plants as a means of self-treatment against coronavirus as they can be toxic. However, the findings could provide a new avenue to develop pharmaceutical treatments for COVID-19.
A new COVID-19 vaccine developed by researchers at the CNB-CSIC appears to protect against infection of the brain and neurological symptoms associated with coronavirus.
COVID-19 infection activates the same inflammatory response in the brain as Parkinson's disease, a new study reports.
SARS_CoV_2, the virus responsible for COVID-19 infects and replicates in astrocytes, reducing neural viability.
The compound Salen effectively binds to a number of SARS_CoV_2, the virus that causes COVID-19, proteins. The findings pave the way to developing new therapeutics to fight coronavirus.
Researchers identify the exertion level where aerosol particle emission increases exponentially, offering an explanation as to why exercise intensity may be linked to the transmission of infections.
Researchers have identified elevated levels of a biomarker in the blood that persists for months in long COVID patients who experience neuropsychiatric symptoms.
Anosmia, the loss of the sense of smell which is a common symptom of COVID-19, may be a secondary consequence of immune system inflammation rather than a direct action of the virus.
COVID-19 infection leaves a gene expression signature in the dorsal root ganglia which persist after the virus has cleared. The signature matched other gene expression patterns seen in pain caused by other conditions.
Study reveals, in detail, how COVID-19 infection affects the central nervous system, including inducing brain injury and inflammation consistent with reduced cerebral blood flow.
BGE-175, an oral drug that reverses multiple aspects of immune aging effectively prevents death in mouse models of COVID-19.
A newly created stem cell model demonstrates a potential route of entry of the COVID-19 virus, SARS_CoV_2, into the human brain.