Post-mortem studies of COVID-19 patients revealed significant signs of neuroinflammation and impaired brain circuits which researchers believe were caused by the disease. Researchers said the changes noticed in the brains of COVID patients were similar to the changes that occur in both Alzheimer's and Parkinson's disease.
Researchers propose a strategy for repurposing geroprotectors, including rapamycin and metformin, as potential COVID-19 infection prevention methods.
BGE-175, an oral drug that reverses multiple aspects of immune aging effectively prevents death in mouse models of COVID-19.
Researchers have identified elevated levels of a biomarker in the blood that persists for months in long COVID patients who experience neuropsychiatric symptoms.
Researchers identify the exertion level where aerosol particle emission increases exponentially, offering an explanation as to why exercise intensity may be linked to the transmission of infections.
Study reveals, in detail, how COVID-19 infection affects the central nervous system, including inducing brain injury and inflammation consistent with reduced cerebral blood flow.
Using over a century of data from other pandemics, and applying knowledge about the current COVID-19 infection, researchers predict the long term effects coronavirus will have on the brain and nervous system.
Combining artificial intelligence technology with data sets related to both Alzheimer's and COVID-19, researchers were able to identify a mechanism by which coronavirus can lead to Alzheimer's-like symptoms. The findings add to the growing body of evidence that COVID-19 infection can have lasting effects on brain function.
Researchers have isolated a set of antibodies from a llama which show promise for the treatment of COVID-19. The NIH-CoVnB-112 nanoantibody bound to ACE2 receptors up to ten times stronger than other lab-produced antibodies. The nanoantibody stuck directly to the ACE2 receptor binding portion of the SARS_CoV-2 spike protein. The protein could be effective in preventing coronavirus infection.
A newly created stem cell model demonstrates a potential route of entry of the COVID-19 virus, SARS_CoV_2, into the human brain.
SARS_CoV_2, the virus responsible for COVID-19, can directly enter the nervous system. The virus can infect the brain, causing alterations in blood vessels and directly disrupt oxygen supply to the organ.
Many people reported they felt like time was passing differently during the initial COVID-19 lockdowns. Researchers explore how the pandemic has contributed to the feeling of time distortion.