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SARS-CoV-2

·October 14, 2024·6 min read

Persistent Virus May Drive Long COVID

A study found people with long COVID symptoms were twice as likely to have lingering SARS-CoV-2 proteins in their blood, suggesting a persistent viral reservoir may drive some ongoing symptoms. Researchers analyzed blood from 706 individuals and found that 43% of those with multisystem long COVID symptoms had viral proteins present months post-infection, compared to 21% of those without symptoms. This discovery hints at potential benefits from antiviral treatments for those with persistent symptoms. However, the study also reveals that not all long COVID cases show persistent viral proteins, indicating other causes may contribute to this complex condition.

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Featured Neurology Neuroscience

·April 27, 2022·6 min read

How Effects on the Brain Can Produce Long COVID

Immune system mediated injury rather than the virus entering and killing brain cells may explain why people experience long-term consequences associated with COVID-19 infection.

Featured Neurology Neuroscience

·December 14, 2021·3 min read

COVID Protein Interacts With Parkinson’s Protein, Promotes Amyloid Formation

The COVID causing SARS-CoV-2 protein interacts with alpha-synuclein, speeding up the formation of amyloid plaques, a new study reports.

Featured Neurology Neuroscience Open Neuroscience Articles

·October 19, 2021·4 min read

Viral Infections Could Promote Neurodegeneration

Some viral infections could increase intercellular spreading of protein aggregates associated with neurodegenerative disorders, increasing the risk for developing Alzheimer's, Parkinson's, and other neurodegenerative diseases.

This shows a man in a facemask surrounded by representations of covid-19

Featured Genetics Neurology Neuroscience

·February 4, 2021·4 min read

The Link Between ‘Alzheimer’s Gene’ and COVID-19

ApoE4, a gene associated with an increased risk of Alzheimer's disease, also appears to increase susceptibility and the severity of COVID-19. SARS-CoV-2, the virus responsible for coronavirus, increased susceptibility to COVID-19 in ApoE4 neurons and astrocytes in brain organoid models.

Featured Neuroscience

·January 28, 2021·6 min read

Melatonin Produced in the Lungs Prevents Infection by COVID-19

Melatonin produced in the lungs acts as a barrier defense against SARS-CoV-2, the virus that causes COVID-19, by blocking the expression of genes that encode proteins in cells acting as viral entry points.

This shows the outline of a brain and head with coronavirus dotted throughout

Featured Neurology Neuroscience

·January 22, 2021·4 min read

COVID-19 Attack On Brain, Not Lungs, Triggers Severe Disease In Mice

Mice exposed to COVID-19 through the nasal passage, researchers noted a rapid and escalated attack on the brain by the virus that triggered a more severe outcome of the infection, even after the lungs were cleared of the disease. Researchers also found virus levels were over 1,000 times higher in the brain than other parts of the body.

Featured Neurology Neuroscience

·December 17, 2020·4 min read

COVID-19 Virus Enters the Brain

The spike protein of SARS-CoV-2 can pass through the blood-brain barrier of mice. The protein likely causes the brain to release cytokines and spark neuroinflammation. The findings add to growing evidence that COVID-19 can enter the brain of those infected by the virus.

Featured Neurology Neuroscience Videos Pain

·October 4, 2020·4 min read

Pain Relief Caused by SARS-CoV-2 Infection May Help Explain Coronavirus Spread

SARS-CoV-2, the virus responsible for COVID-19 suppresses pain. Findings suggest the pain suppression caused by the infection may be responsible for viral spread, as people do not feel as ill as they actually are.

Featured Neuroscience Open Neuroscience Articles

·September 28, 2020·5 min read

COVID-19 May Deplete Testosterone

A new study suggests COVID-19 may deplete testosterone levels in males. Findings reveal as testosterone decreased, the severity of coronavirus increased. Men who died from coronavirus infection had significantly lower mean testosterone than those who recovered. For those who were asymptomatic, 65.2% reported a loss in libido.

Featured Open Neuroscience Articles Psychology

·September 14, 2020·4 min read

Substance Use Disorders Linked to COVID-19 Susceptibility

People with a history of substance use disorder are more susceptible to COVID-19 infection and have an increased risk of experiencing worse outcomes, a new study reports.

Featured Neurology Open Neuroscience Articles Visual Neuroscience

·September 6, 2020·5 min read

Post-COVID Syndrome Severely Damages Children’s Hearts

Multisystem inflammatory syndrome in children, a Kawasaki-like disease that appears to result from COVID-19 infections, damages the heart to such an extent that children will need lifelong monitoring and interventions.

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SARS-CoV-2

Persistent Virus May Drive Long COVID

How Effects on the Brain Can Produce Long COVID

COVID Protein Interacts With Parkinson’s Protein, Promotes Amyloid Formation

Viral Infections Could Promote Neurodegeneration

The Link Between ‘Alzheimer’s Gene’ and COVID-19

Melatonin Produced in the Lungs Prevents Infection by COVID-19

COVID-19 Attack On Brain, Not Lungs, Triggers Severe Disease In Mice

COVID-19 Virus Enters the Brain

Pain Relief Caused by SARS-CoV-2 Infection May Help Explain Coronavirus Spread

COVID-19 May Deplete Testosterone

Substance Use Disorders Linked to COVID-19 Susceptibility

Post-COVID Syndrome Severely Damages Children’s Hearts

Newly Mapped Brain Circuit Reveals How Memories Stay Stable During Learning

Common Antidepressant Eases Core Symptoms Within Two Weeks

Toddler Gut Bacteria Linked to Childhood Anxiety and Depression

$2 Saliva Test Could Help Detect Depression and Schizophrenia

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