Researchers report the injury pathway is triggered when Unc-104 is mutated. This mutation leads synapses to become defective.
Researchers have discovered how to reverse abnormal axonal development.
Incorrect levels of GSK-3ß, a protein linked to Alzheimer's disease, can lead to neural blockages.
Researchers question why numerous different RNAs are transported to synapses. One reason, they suggest, is that they are stored to later help maintain long-term memory.
