Chronic stress accelerates the body's epigenetic clock, however, those that can manage the effects of stress by strengthening their emotional regulation and self-control can slow the process.
Amyloid-beta accumulation in the brain may contribute to deficits in circadian regulation of learning and memory during the early stages of Alzheimer's disease.
Researchers have identified a new gene called Tango10 which plays a key role in daily circadian behavioral rhythms.
More time spent outside in natural light was associated with improved mood, better sleep quality, and ease of waking.
A reconstituted circadian clock of cyanobacteria can run for consistent days, allowing researchers to study the interactions of the clock proteins in real-time and observe how the clock exerts control over gene expression.
A new study traces the mechanisms that link environmental signals and our circadian clocks.
Daily release of glucocorticoids depends on coordination between the clock gene and activity rhythms of neurons within two parts of the hypothalamus.
Long-term circadian rhythm disruptions induce Alzheimer's disease-like pathology in rats, which can be reversed by administering fluoxetine. Additionally, elevated levels of amyloid beta and circadian rhythm disruptions can trigger each other, leading to the cascade of neurological symptoms of dementia.
People who are genetically predisposed to be "early birds" only take a few days to adjust to time changes as a result of daylight savings. Night owls can take over a week to adjust to the time change.