Sleep disruptions caused by daylight savings time can lead to a range of health-related problems. Researchers provide tips on how to adjust your sleep patterns to compensate for daylight savings.
Our bodies can predict the timing of regular meals, a new study reports. Additionally, a person's daily blood-glucose rhythms may be driven by meal size in addition to meal time.
REM sleep is 30 minutes longer in Winter than in Summer for most people, a new study reveals.
Researchers say that late-morning exercise may be more effective in boosting metabolism and burning fat than late-evening exercise. The findings could prove valuable to those who are overweight and looking to lose fat by implementing an exercise program.
Sleep intervention programs for adults on the autism spectrum show promising results in reducing insomnia and co-occurring anxiety symptoms.
Postmortem brains of those with schizophrenia have fewer genes associated with 12-hour activity cycles in the dorsolateral prefrontal cortex. Mitochondrial-related genes in the dlPFC did maintain a 12-hour rhythm, but their activity did not peak at normal times.
Neural networks play a key role in regulating circadian rhythms through the mediation of cAMP. The findings may help with developing new strategies to manage circadian rhythm problems such as sleep disorders.
Time-restricted eating influences gene expression over more than 22 regions in the brain and body. The findings have implications for a range of health disorders in which time-restricted eating appears to have potential benefits.
Those who exercise either early or late in the morning were 11% and 16% respectively at a lower risk of coronary artery disease. Those who exercise later in the morning were 17% less likely to have a stroke than those in the control group.
Contrary to previous findings, a new study reports those who rise early tend to have superior verbal skills compared to night owls.
Researchers reveal how night owls can turn themselves into early rising early birds.
Researchers identified a molecularly-defined cell population in the hypothalamus that causes a shift in the sleep-wake cycle as a result of exposure to psychostimulants.
