The severity of Charcot-Marie-Tooth disease is linked to the degree to which the aminoacyl-tRNA synthetases enzyme is relaxed or extended.
Increasing levels of the MFN1 protein counterbalances mutated MFN2, reducing symptoms associated with Charcot-Marie-Tooth disease and neurodegeneration in mouse models.
New research could lead to the development of treatments to correct mitochondrial dysfunction associated with Charcot Marie Tooth disease.
Researchers report on how Schwann cells transform to play a more active role in neurogenesis following nerve injury.
Researchers believe the subject of a world famous painting may have suffered from Charcot-Marie-Tooth Disease.
Researchers discover a roundworm contains two proteins that play a role in the degeneration of axons.
A new study reveals how Schwann cells in the PNS degrade myelin following nerve injury.
Researchers uncover a key to the development of the peripheral nervous system, providing new hope to patients suffering from hereditary neuropathy.
