A new mouse study reveals that exposure to BPA at levels 25 times lower than deemed safe has an impact on brain development.
Exposure to BPA during the gestational period leads to decreased neural viability and neural density in the hippocampus of male offspring. Additionally, exposure led to the dysregulation of ASD-related genes in the hippocampus. Findings suggest BPA may serve as an environmental factor that contributes to the prevalence of male-bias in autism.
A new method reveals BPA levels may be more than 44 times higher in humans than previously believed.
Exposure to BPA appears to have a transgenerational effect on autism risk. Mice whose great grandmothers were exposed to BPA during pregnancy exhibited social behavioral deficits associated with ASD.
According to researchers, even lower levels of BPA consumed during pregnancy can lead to neurodevelopmental problems and affected brain function later in life for offspring.
BPS affects a neuroanatomy and maternal behavior in pregnant and lactating mice, including an association with an increased risk of infanticide.
New research suggests environmental exposure to bisphenol A (BPA), a chemical commonly found in plastics and resins, could suppress a gene critical to nerve cell function and the development of the central nervous system. Exposure to BPA could predispose humans to a number of neurodevelopmental and other health disorders.