Researchers have identified a molecule in the blood that is produced during exercise. The molecule, Lac-Phe, can effectively reduce food intake and obesity in mouse models.
In the wake of the school shooting in Uvalde, psychologists report on the best way to address such tragedies to young children and explore how to help teachers and those directly affected by the events.
Hyperactivity in a serotonin-dopamine circuit in the brain appears to be responsible for persistent anorexia in animal models. The DRD1 receptor appears to drive the hyperactivity. Deleting the DRD1 gene restored normal eating behaviors in animals.
Type 1 interferon is a major diver of cognitive impairment and memory loss in mouse models of Alzheimer's disease. Blocking IFN reversed both cognitive impairments and memory deficits in the animal models.
Oleic acid produced in the brain is an essential regulator of processes that enable memory, learning, and mood regulation. Oleic acid, which is abundant in olive oil, also promoted neurogenesis and increases cell proliferation.
Electrical stimulation improves verbal memory for those with responsive neurostimulation implants when activated during certain memory tasks.
Mouse study reveals brain activity patterns associated with memory recall become active when learning by observation.
In fruit fly models, an ABCA1 agonist can restore specific alterations of neuroprotective mechanisms associated with a genetic predisposition to Alzheimer's disease.
A new computational method sheds light on the intricacies of brain structure and function.
Researchers discuss how to accommodate family members with dementia over the holidays to make Christmas a meaningful and joyous event for the entire family.
Combining deep-brain stimulation with intracranial EEG, researchers achieved an individualized understanding of specific brain networks that contributed to an individual's depression symptoms and identified stimulation patterns best suited to each patient for symptom relief.
Restoring normal sleep patterns by activating the thalamic reticular nucleus with chemogenetics reduced the accumulation of amyloid-beta plaques in the brains.
