During periods of social isolation, astrocytes in the brain become hyperactive. This suppresses circuit formation and memory formation. Reversing astrocyte hyperactivity can help mitigate memory deficits associated with social isolation.
Researchers develop a novel tool that allows for the study of the communication of microbes in the gastrointestinal tract and the brain.
Mutations in the gene for the serotonin 2C receptor play a key role in obesity and dysfunctional behaviors in both human and animal models.
In the olfactory bulb, levels of oxytocin rise and peak when new neurons incorporate themselves into neural networks. The findings shed new light on adult neurogenesis and brain plasticity.
Exercise can help improve mood and overall symptoms of those who suffer from seasonal affective disorder (SAD).
Study reveals how changes in pupil size affect the way we perceive our surroundings.
Obesity is, in part, determined by epigenetic development in the arcuate nucleus of the hypothalamus. Findings suggest developmental epigenetics plays a significant role in both environmental and genetic influences on obesity risk.
Researchers have developed a new sensor that allows scientists to image the brain without missing signals for an extended period of time and deeper in the brain than current technology allows.
Researchers report they have identified the first natural light-gated potassium channel-rhodopsins.
Researchers have identified a molecule in the blood that is produced during exercise. The molecule, Lac-Phe, can effectively reduce food intake and obesity in mouse models.
In the wake of the school shooting in Uvalde, psychologists report on the best way to address such tragedies to young children and explore how to help teachers and those directly affected by the events.
Hyperactivity in a serotonin-dopamine circuit in the brain appears to be responsible for persistent anorexia in animal models. The DRD1 receptor appears to drive the hyperactivity. Deleting the DRD1 gene restored normal eating behaviors in animals.