The stroke byproduct acrolein activates the precursor of heparanase, an enzyme that degrades the glycocalyx in the blood-brain barrier.
An antibody called HAE-4 targets APOE and removes Alzheimer's related amyloid plaques in mouse models. The antibody improves blood vessel function in the brain without raising the risk of brain bleeds.
The TPS neural stimulation technique is able to non-invasively target and activate deep brain regions, providing a new treatment potential for a range of neurological and psychological disorders.
Treatment with LAU-0901, a synthetic molecule that blocks pro-inflammatory platelet-activating factor, in addition to aspirin-triggered NPD1, reduced the size of damage areas in the brain, initiated repair mechanisms, and improved behavioral recovery following ischemic stroke.
Repetitive transcranial magnetic stimulation significantly decreases post-stroke depression by increasing brain activity.
The risk of stroke is 54% higher in those who experience symptoms of depression, a news study reports.
Direct carotid puncture is a safe and effective alternative to thrombectomy for stroke patients.
A combination of the Zika virus and Chikungunya, two mosquito-borne infections, increase stroke risk, a new study reports.
Blood transfusions lead to improved outcomes in mouse models of ischemic stroke. Researchers believe the findings could be used for the treatment of stroke in humans.
A new online tool can help individuals determine their risk of ischemic stroke. Preliminary findings from those who used the tool report a strong association between metabolic syndrome and ischemic stroke risk was in white women.
DHA treatment reduced the size of the damaged brain area and initiated a repair mechanism in animal models of stroke. DHA affected the levels of MANF and TREM2, two proteins critical for communication between brain cells.
Coronavirus infection can lead to an array of neurological complications, including delirium, stroke, and a rare, often fatal neuroinflammatory condition called ADEM. The neurological complications did not always correlate with the severity of respiratory symptoms. In some patients, brain inflammation was likely caused by an immune response to COVID-19, suggesting the neurological damage may be a result of immune system activation rather than the virus itself.
