Switching to a diet that includes more leafy greens, including spinach and kale, reduces inflammation and oxidative stress, and improved symptoms associated with chronic migraine.
Post-mortem studies of brain tissue from ALS patients reveal an abnormal form of tau is present in novel brain areas, and the tau interacts with DRP1. The tau appears to cause the brain cell's mitochondria to fragment and increase oxidative stress. Reducing tau reversed the effect, decreasing oxidative stress and mitochondrial fragmentation.
A new study reveals how adipocytes, or fat cells, play a role in cognitive decline and increased neurodegeneration risks. Using genetically modified mouse models, researchers found oxidative stress through exposure to a Western diet increased the production of inflammatory cytokines confined to adipocytes and altered protein markers of memory and cognition in the hippocampus.
A single time vaping increases oxidative stress levels up to four times in non-smokers.
Study identifies specific chemical features of tau that may cause it to accumulate in the brain and trigger Alzheimer's disease.
CRISPR gene-editing technology allowed researchers to pinpoint specific neurons and mechanisms related to the effects of oxidative stress in the brain.
From impairing nervous system function and changing bacterial communities in the microbiome to increasing oxidative stress and inflammation, researchers report on the ways in which exposure to chemical pollution harms the body.
Free radicals help control cellular processes vital for neuroplasticity and neurogenesis in mice.
An examination of brainstems from children and young adults constantly exposed to air pollution reveals markers of Alzheimer's disease, Parkinson's disease, and motor neuron disease. Findings suggest air pollution poses risks of serious neurological damage from an early age.
Death caused as a result of sleep deprivation is preceded by the accumulation of reactive oxidative species in the gut.
Regular exercise may help prevent or reduce the severity of ARDS for those with coronavirus. A single session of exercise increases the production of a critical antioxidant, called EcSOD, helping to reduce the effects of COVID-19 infection.
A new method called Tox-seq found only one sub-group of microglia caused oxidative stress. The gene expression signatures of this sub-group from a mouse model matched patterns observed in cells suspected of causing damage in progressive multiple sclerosis patients. The pattern of gene expression associated with oxidative stress in mouse cells included genes involved in coagulation.
