Data collected from a tetraplegic man learning a BCI to play Simon showed the brain replays the learned information during sleep.
People with primary progressive aphasia who have difficulty producing complex words as a result of their condition, compensate by stringing together a sequence of simpler words to convey the idea they wish to express.
Exposure to the opioid fentanyl induces autism-like behaviors in mice by activating mu-opioid receptors in the anterior cingulate cortex.
Using psychiatric medications to treat mental health disorders in children and adolescents does not increase the risk of developing substance use disorders later in life. In fact, pharmacological interventions reduce the risk of developing SUD for those with MDD, ADHD, and psychotic disorders.
The accumulation of amyloid-beta and tau proteins disrupts the connections between brain regions associated with memory years before symptoms of Alzheimer's disease appear.
Neurons in the anterior cingulate store social rank information to inform upcoming decisions. The findings could shed new light on social deficits associated with ASD and schizophrenia.
Researchers report some patients with long-COVID have lasting nerve damage that appears to be caused by infection-triggered immune dysfunction.
Study reports 1 in 5 teens and young adults who seek treatment for alcohol and substance use disorders may have previously unrecognized social impairments characteristic of ASD.
A new study reports people with depression are 2.2 times more likely to endorse misinformation about the COVID-19 vaccine, and 2.7 times more likely to be vaccine-resistant. Additionally, rates of depression are three times higher than before the pandemic began.
Inhibition of the OR5B21 olfactory receptor gene significantly decreased the metastasis of breast cancer cells to the brain, lungs, and bones, researchers report.
Study reveals there are no net benefits of omega-3 supplementation for preventing depression or boosting mood.
Post-mortem studies of brain tissue from ALS patients reveal an abnormal form of tau is present in novel brain areas, and the tau interacts with DRP1. The tau appears to cause the brain cell's mitochondria to fragment and increase oxidative stress. Reducing tau reversed the effect, decreasing oxidative stress and mitochondrial fragmentation.
