Capsular polysaccharide A (PSA), an envelope molecule, may help to boost the immune system and protect against potentially fatal neuroinflammation associated with Herpes Simplex Encephalitis (HSE). Mice given PSA survived exposure to a lethal herpes simplex viral infection, while those not treated with the probiotic did not, despite both groups being treated with a common antiviral used to treat HSE.
A new study reports herpes virus utilizes ancient RNA to proliferate, mimicking the same process tumors have been found to manipulate. The findings could have implications for new treatment options and also may shed light on neurodegenerative diseases.
A new study reports people with schizophrenia have higher levels of antibodies against Epstein-Barr virus, a herpes virus that causes mononucleosis. Researchers propose two explanations for the link to the heightened immune response to the virus: schizophrenia may alter the immune system, making patients more susceptible to EBV, or EBV may increase the risk of developing schizophrenia.
A new study looks at the possible causal link between the herpes virus and Alzheimer's disease. Researchers report antiviral drugs significantly reduce the risk of developing dementia in patients with severe herpes infections.
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Researchers provide evidence that certain species of human herpesvirus contribute to the development of Alzheimer's disease. The study reports high levels of human herpesvirus 6A and 7 were found in brain samples showing signs of Alzheimer's neuropathology. The findings offer hints of the viral mechanisms that could trigger or exacerbate AD.
A new study in Scientific Reports concludes human herpesvirus 6 may impair the brain's ability to repair itself in demylination diseases, such as multiple sclerosis. HHV-6 is the most common human herpes virus, with an estimated 80% of people being exposed to infection during childhood. Researchers found the virus produces a protein that can impair the ability of brain cells to repair damaged myelin.
According to researchers, the expression of human amyloid beta protected against potentially lethal infections in mice, roundworms and cultured human brain cells.
A new study reports certain strains of the herpes virus are able to infect neurons and could underlie some symptoms of neurological disorders such as MS.