Deep brain stimulation of the nucleus accumbens helps control symptoms of binge eating disorder and assists in weight loss.
Young people who consume "cheat meals", or meals that break a normal diet as a treat, are more likely to suffer from eating disorders, a new study reports.
Neuroimaging study reveals those with anorexia have noticeable reductions in cortical thickness, subcortical volume, and cortical surface area. The reductions are between 2 to 4 times larger than abnormalities in brain size and shape in those with other mental illnesses.
While there's nothing wrong with having high personal standards, perfectionists often feel as though their achievements and successes are never good enough. Perfectionism increases the risk of depression, anxiety, eating disorders, and several other mental health disorders.
Hyperactivity in a serotonin-dopamine circuit in the brain appears to be responsible for persistent anorexia in animal models. The DRD1 receptor appears to drive the hyperactivity. Deleting the DRD1 gene restored normal eating behaviors in animals.
Children with binge eating disorders have differences in gray matter density compared to their peers who do not experience overeating disorders.
A lifetime history of APEDS use is associated with eating disorder symptoms, specifically when using protein and creatine supplements, or diuretics.
Eating disorder behaviors are reinforced due to changes in the brain's reward response processes and alterations in the food intake control network.
Activity in the dorsal mid insular could drive different interpretations in bodily sensations in those with mental health disorders like depression, anorexia, and panic disorders.
Children aged between 7 and 9 who experience frequent abdominal pain are more likely to develop fasting habits and eating disorders to help control their weight by age 16.