Caspase-2 is a key regulator of a signaling pathway which leads to cognitive decline in Alzheimer's disease, a new study finds.
Researchers suggest lifetime brain growth and early mental stimulation independently help protect against cognitive decline in multiple sclerosis.
Researchers discover by looking at amyloid-beta and tau independently that the interaction between the proteins could be the key to the cognitive decline associated with Alzheimer's disease. As the interaction between the two proteins increased, the progression of Alzheimer's worsened.
Researchers show memory pathology in older mice can be reversed by blocking the activity of a specific receptor in the brain. Blocking the B1R receptor helps recover memory and cerebrovascular function.
New research finds an increased risk for cognitive decline in patients with cardiovascular disease and type 2 diabetes.
Using MRI neuroimaging technology, researchers were able to predict the risk for cerebrovascular disease in patients.
A new study found soldiers returning from Iraq and Afghanistan with mild TBI have measurable abnormalities in white matter when compared to returning veterans who have not experienced TBI. These abnormalities appear to be related to the severity of the injury and are related to cognitive deficits.
Scientists have discovered that significantly more neurons are generated in the brains of older animals if the signaling molecule Dickkopf-1 is turned off. The results give rise to the question whether the function of Dickkopf-1 may be turned off using drugs to prevent age related cognitive decline.
When the brain produces too much nitric oxide, it contributes to the severity and progression of stroke and neurodegenerative diseases such as Alzheimer’s. Researchers recently discovered that nitric oxide not only damages neurons, it also shuts down the brain’s repair mechanisms.
A new research study conducted has found that the herbal supplement Ginkgo biloba does not improve cognitive function in patients with Multiple Sclerosis (MS.)
Penn Medicine research presented today at the 2012 Alzheimer's Association International Conference shows that an anti-tau treatment called epithilone D (EpoD) was effective in preventing and intervening the progress of Alzheimer's disease in animal models, improving neuron function and cognition, as well as decreasing tau pathology.
A diet lacking in omega-3 fatty acids, nutrients commonly found in fish, may cause your brain to age faster and...
