While commonly implicated in long-term memory, researchers report the hippocampus plays a critical role in short-term memory and decision making.
Aphantasia is marked by the inability to generate visual images in the mind's eye. Researchers explore the neurobiological basis for the disorder.
As novel visual patterns become familiar, stark changes occur in the visual cortex. Gamma rhythms give way to low-wave beta waves, and the neural activity switches from PV neurons in favor of inhibitory SOM neurons.
New research indicates the existence of an unconscious iconic memory store that supports predictions made by the global workspace theory of consciousness. It also shows that visual masking does not erase memory traces of masked stimuli but only limits conscious access.
In visual working memory, the brain immediately transfers the memory of object shifts by re-encoding the memory among neurons in the opposite brain hemisphere.
While people with aphantasia lack visual imagery ability, they have intact spatial memory. Findings suggest mental imagery recall and spatial memory may be stored differently in the brain.
Objects are not only represented in one form of short term memory but in several forms simultaneously.
Brain activity in the fusiform face area on the right side of the brain showed no difference in those with face blindness compared to those without the condition. However, researchers found those with prosopagnosia had reduced activity in a corresponding area on the left side of the brain.