Researchers discovered a correlation between obesity-related neurodegeneration and Alzheimer's disease pathology. Losing weight, they say, can slow age-related cognitive decline and reduce the risk of developing Alzheimer's.
Study reports in the short term, astrocytes regulate caloric intake by controlling the signaling pathway between the gut and brain. Eating high-fat or high-calorie diets disrupts this pathway.
High-fat diets promote early inflammatory responses in the brain via an immune pathway associated with diabetes and neurodegenerative diseases. The findings suggest a link between metabolic dysfunction and cognitive impairment.
Mutations in the gene for the serotonin 2C receptor play a key role in obesity and dysfunctional behaviors in both human and animal models.
Physically active, married females who are from financially stable backgrounds, are not obese, and do not suffer from insomnia are more likely to maintain good health and less likely to suffer cognitive, physical, or emotional problems as they age.
Those who report trouble sleeping are at increased risk of poor cardiometabolic health problems which can lead to type 2 diabetes.
People who ate almonds lowered their energy intake by 300 kilojoules at their following meal. Almonds alter appetite-regulating hormones and help to reduce food intake.
High-fat diets induce hyperalgesic priming, a neurological change that represents the transition from acute to chronic pain, and allodynia or pain resulting from stimuli that do not normally provoke pain.
Stress impacts the brain's response to food, researchers report. Additionally, both lean and obese people react to food cues in brain areas associated with reward and cognitive control.