Neurons in the memory-associated entorhinal cortex of super-agers are significantly larger than their cognitively average peers, those with MCI, and even in people up to 30 years younger. Additionally, these neurons contained no signs of Tau, a hallmark of Alzheimer's disease.
The formation of long-term memory relies on intrinsic neurological learning processes and the repetition of events.
Assessing a child's exposure to air pollution on a monthly basis from conception to the age of 8.5, researchers found the greater the exposure to air pollution before the age of 5, the greater the alteration in brain structure by preadolescence.
COVID-19 infection has been linked to a range of lasting neurological and psychological disorders, including depression, memory problems, and Parkinson's-like disorders, within the first year following infection.
In mice genetically more susceptible to PTSD following a stressful event, researchers found an increased expression of cortisol receptors on neurons in the CA1 region of the dorsal hippocampus. Those increased receptors enabled an elevated expression of the HCN1 protein and TRIP8b, reducing neural excitability.
Study reveals a neural signal in the hippocampus that enables the brain to alternate between learning and remembering modes.
Study found no correlation to substantiate the claim that better autobiographical memory enhances creativity or creative thinking.
Researchers have identified a new mechanism within neurons that cause memories associated with social interactions to decline with age. Additionally, they were able to reverse the memory loss in mouse models.
Super recognizers focus less on the eye region and distribute their gaze more evenly than typical viewers, extracting more information from other facial features.