Anticoagulant drugs that inhibit thrombin were shown to reduce the number of brain metastases in mouse models.
Heating up cancer cells as they are being targeted with chemotherapy appears to be a highly effective way of killing them off.
Study reports brain tumors may arise when damaged brain tissue does not heal correctly. Researchers say some glioblastoma form when the normal healing process gets derailed by mutations. This process could begin many years before patients become symptomatic of brain cancer.
Loperamide, an anti-diarrhea medication, induces autophagy in glioblastoma brain cancer cells.
Researchers have identified a cancer-causing mutation in the PDGFRA gene that drives cell mutation and growth when activated. The findings have implications for the treatment of a subset of glioblastoma brain cancer.
Reductive stress promotes protein aggregation in neuroblastoma cells and impairs neurogenesis.
Increased YTHDF3 expression appears to be a key driver in brain metastases.
Myeloid cells can suppress the immune response, allowing breast cancer cells to metastasize in the brain and form secondary tumor sites.
Jet lag alters the microenvironment around cancerous tumor cells, making it more favorable for tumors to grow, and hinders the body's natural immune response.
A new computational model predicts how information deep inside the brain could flow from one network to another, and how neural network clusters can self optimize over time.
MP-Pt(IV), a second generation prodrug appears to have curative properties against glioblastoma when coupled with chemotherapy in mouse models.
Study reveals how two key molecules, Rab27b, and epiregulin, interact to contribute to radioresistance in glioblastoma brain cancer.
