Targeting the GAT3 protein in the thalamus could help block and prevent long-term damage following brain injury.
Study reveals how neurons and glial cells contribute to drive neurodegeneration following brain injury.
College football players are 5 times more likely to report cognitive impairment, 2.5 times more likely to experience recurrent headaches, and 65% more likely to have cardiovascular problems in their lifetime than their non-football playing peers. Additionally, mortality from brain and other nervous system cancers was 4 times higher in former college football players than the general population.
Neuroimaging study revealed a significant number of professional rugby players had white matter abnormalities and abnormal changes to white matter volume over time.
Damage to highly connected regions of white matter in the brain following injury is more predictive of cognitive impairment than damage to highly connected gray matter hubs.
Study reveals there is no significant uptick in men who played high school football reporting problems with brain health in middle age compared to their peers who did not play sports. However, ex-football players were more likely to experience sleep problems and be prescribed medications for chronic pain during mid-life.
In veterans who suffered a TBI, lack of sleep was associated with enlarged perivascular spaces and an increase in post-concussive symptoms.
Pomegranate juice appears to have neuroprotective effects in pregnancies marked by intrauterine growth restriction. Researchers found pomegranate juice reduced the risk of brain injury in infants with IUGR, especially when pregnant women drank it during the third trimester.
Using an egg, researchers discover how trauma occurs to the brain as a result of impact.
Myelomonocytic cells, a type of immune cell, can both harm and help the brain following injury.
Response to speech can be measured with EEG in unresponsive patients with TBI. The strength of the response can be used to accurately make a prognosis.
Smaller brain volumes in the cingulate cortex, insular, and superior frontal cortex predict an increased likelihood of developing PTSD three months after brain injury.
