Neuroimaging study reveals those with anorexia have noticeable reductions in cortical thickness, subcortical volume, and cortical surface area. The reductions are between 2 to 4 times larger than abnormalities in brain size and shape in those with other mental illnesses.
Hyperactivity in a serotonin-dopamine circuit in the brain appears to be responsible for persistent anorexia in animal models. The DRD1 receptor appears to drive the hyperactivity. Deleting the DRD1 gene restored normal eating behaviors in animals.
Eating disorder behaviors are reinforced due to changes in the brain's reward response processes and alterations in the food intake control network.
Activity in the dorsal mid insular could drive different interpretations in bodily sensations in those with mental health disorders like depression, anorexia, and panic disorders.
Contrary to popular belief, people with eating disorders like bulimia nervosa do not lose control and binge eat in response to stressful events.
People with eating disorders are up to twelve times more likely to experience body dysmorphia than those without a history of eating disorders. 76% of those with eating disorders report suffering from body dysmorphia, a new study reveals.
Study reveals how eating disorders in some women are inextricably linked to their culture and upbringing.
The findings of three new studies reveal only 50% of those with eating disorders seek help for their condition. Certain demographics are less likely to seek help. Those with eating disorders have a 5-6 times higher risk of suicide attempts.