Contact-seeking behavior in mice is driven by amylin peptide in the medial preoptic area of the forebrain.
Researchers have identified a mechanism that drives hunger. A new study reports the sight or smell of food can temporarily turn of AgRP neurons, which drive the urge to eat. These neurons remain inactive until the brain receives a signal from the gut that calories have been consumed.
Researchers report a hormone called amylin acts in the brain to help control food consumption.
A new study suggests amylin could be a second amyloid and a new biomarker for age related dementia and Alzheimer's disease.