Contact-seeking behavior in mice is driven by amylin peptide in the medial preoptic area of the forebrain.
When injected into mouse models of Alzheimer's disease, two short peptides significantly improved memory, reduced inflammation, and lowered amyloid plaque buildup in the brain.
Researchers have identified a mechanism that drives hunger. A new study reports the sight or smell of food can temporarily turn of AgRP neurons, which drive the urge to eat. These neurons remain inactive until the brain receives a signal from the gut that calories have been consumed.
A new study reports a compound by the name of AC253 may inhibit amyloid beta.
Researchers report a hormone called amylin acts in the brain to help control food consumption.
A new study suggests amylin could be a second amyloid and a new biomarker for age related dementia and Alzheimer's disease.
