ALS

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ALS

Featured Neurology Neuroscience Open Neuroscience Articles

·April 20, 2022·7 min read

Study of Former Notre Dame Football Players Finds College Players More Likely to Have Brain Disorders

College football players are 5 times more likely to report cognitive impairment, 2.5 times more likely to experience recurrent headaches, and 65% more likely to have cardiovascular problems in their lifetime than their non-football playing peers. Additionally, mortality from brain and other nervous system cancers was 4 times higher in former college football players than the general population.

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Featured Neuroscience Neurotech

·April 5, 2022·7 min read

System Helps Severely Motor-Impaired Individuals Type More Quickly and Accurately

Nomon, a newly designed flexible system, incorporates probabilistic reasoning to learn how users with motor impairments and paralysis make selections when typing and adjust the interface to improve speed and accuracy.

Featured Genetics Neurology Neuroscience Neuroscience Videos

·April 3, 2022·5 min read

Mutations in Noncoding DNA Are Found to Protect the Brain From ALS

Mutations in the IL18RAP gene reduce inflammation and appears to protect the brain against ALS.

Featured Neurology Neuroscience Open Neuroscience Articles

·March 18, 2022·5 min read

Could There Be a Link Between the Enteric Neurons, Gut Microbiome and ALS?

Study reveals an association between intestinal inflammation and the gut microbiome in the development and progression of ALS.

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Featured Neurology Neuroscience

·March 10, 2022·4 min read

Motor Neuron Toxin Associated With ALS Identified

PolyP, an inorganic polyphosphate released by astrocytes in people with ALS and frontotemporal dementia contributes to the signature motor neuron death associated with the disease pathologies.

Featured Genetics Neurology Neuroscience

·February 28, 2022·4 min read

A New Molecular Pathway Shared by Two Neurodegenerative Disorders Discovered

Mislocalization of the TDP-43 protein alters the genetic instructions for UNC13A. The findings provide a potential new therapeutic target for the treatment of ALS and frontotemporal dementia.

Featured Neurology Neuroscience Open Neuroscience Articles

·January 28, 2022·4 min read

Simple and Reliable ALS Diagnosis With Blood Tests

Measuring the level of neurofilaments in the blood may be a reliable biomarker for the early diagnosis of ALS.

Featured Genetics Neurology Neuroscience

·January 19, 2022·6 min read

Astrocyte Studies Reveal Harmful Changes in Amyotrophic Lateral Sclerosis

In patients with ALS, astrocytes within the brain become pro-inflammatory and tend to lose their protective function, resulting in changes in the ability to uptake glutamate.

Featured Genetics Neurology Neuroscience

·January 9, 2022·3 min read

Research Clears up How Brain ‘Cleaners’ Fail in ALS

Findings point to microglial TREM2 as a potential target for the treatment of ALS.

Featured Genetics Neurology Neuroscience

·November 29, 2021·5 min read

Why Some People Experience Early Onset Neurodegenerative Diseases

A new study sheds light on the genetic causes of a range of neurodegenerative disorders, including ALS, Parkinson's disease, and Huntington's disease, and determines factors that impact the age of onset as well as disease severity.

Featured Neurology Neuroscience

·November 15, 2021·6 min read

Altered Fat Metabolism Plays Key Role in ALS

People with ALS have 2.5-fold higher levels of arachidonic acid, a lipid commonly found in fatty parts of meat and fish that spurs on inflammatory process, in their spinal motor cells than people without the disease. Treatment with caffeic acid, an anti-inflammatory compound naturally found in coffee, tea, and tomatoes, reduced some of the symptoms associated with ALS, and extended lifespan in animal models.

Featured Neurology Neuroscience

·November 10, 2021·4 min read

New Insights on ALS May Point To a Potentially Promising Treatment Strategy

Post-mortem studies of brain tissue from ALS patients reveal an abnormal form of tau is present in novel brain areas, and the tau interacts with DRP1. The tau appears to cause the brain cell's mitochondria to fragment and increase oxidative stress. Reducing tau reversed the effect, decreasing oxidative stress and mitochondrial fragmentation.

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