In mice, disruption of the growth hormone receptor gene in fat cells improved insulin sensitivity in advanced age and increased lifespan.
Newly discovered sensory neurons send information related to stress and metabolism from adipose fat tissue to the brain.
Researchers have found a direct connection between flavan 3-ols consumption and fat browning by activation in the sympathetic nervous system. The findings could help in the development of new treatments for obesity-related disorders.
Normally bushy networks of neural fibers within fat tissue shrink in the absence of leptin, but grow back when the hormone is administered in drug form. The alterations influence the ability to burn energy stored in fat in mouse models.
Researchers report just one night of sleep loss can have a tissue specific impact on metabolism and the regulation of gene expression. The study could explain why those who suffer chronic sleep loss or work shifts are at greater risk of obesity and type 2 diabetes.
Researchers have identified a population of immune cells that appear to be associated with neurons which play a role in fat storage and obesity.
A new study reveals a molecular process in the brain which impacts how much energy we can burn and how much weight we can lose.
