AgRP neurons in the hypothalamus control the release of endogenous lysophospholipids, helping to control the excitability of cerebral cortex neurons and stimulating the desire for food intake.
Researchers say the sight and smell of food alone may be enough to prompt the liver to start the processes that help digest food.
Researchers have identified a mechanism that drives hunger. A new study reports the sight or smell of food can temporarily turn of AgRP neurons, which drive the urge to eat. These neurons remain inactive until the brain receives a signal from the gut that calories have been consumed.
Researchers report the brain's ability to sense insulin and coordinate feeding with energy expenditure is controlled by a mechanism that is turned on after fasting to inhibit insulin response and conserve energy. After feeding, the mechanism is turned off to facilitate insulin response and expend energy. However, in obese people, researchers believe the switch may stay on all the time.
A new study could help explain how the hungry brain hinders dieting.
New findings revise the current models for homeostatic control, researchers report.
Researchers discover a control mechanism for neurons involved in hunger and eating disorders.