Researchers report glia play a critical role in feeding behavior and appetite control.
The absence of a specific type of neuron in the brain can lead to obesity and diabetes in mice report researchers. The outcome, however, depends on the type of diet that the animals are fed.
Researchers report the brain's ability to sense insulin and coordinate feeding with energy expenditure is controlled by a mechanism that is turned on after fasting to inhibit insulin response and conserve energy. After feeding, the mechanism is turned off to facilitate insulin response and expend energy. However, in obese people, researchers believe the switch may stay on all the time.
Study identifies the neural circuits that cause hunger-induced increases in the preference for sweet foods and decreased sensitivity to aversive tastes.
Researchers discover a control mechanism for neurons involved in hunger and eating disorders.
New findings revise the current models for homeostatic control, researchers report.
A new study could help explain how the hungry brain hinders dieting.
A new gene editing study reveals how the hormone leptin can help to prevent diabetes and obesity.
A new study could have positive implications for developing anti obesity treatments.
Researchers say the sight and smell of food alone may be enough to prompt the liver to start the processes that help digest food.
Researchers have identified a mechanism that drives hunger. A new study reports the sight or smell of food can temporarily turn of AgRP neurons, which drive the urge to eat. These neurons remain inactive until the brain receives a signal from the gut that calories have been consumed.
Induced changes in the function of AgRP neurons may contribute to depression, researchers report.
