Rare variants of the autism-associated ANK2 gene alter the architecture and organization of neurons, potentially contributing to autism and neurodevelopmental comorbidities.
A new study sheds light on the molecular basis of memory. The study details how the binding sites of CamKII act to align actin fragments, creating bundles that support skeletons of dendritic spines.
Severed axonal segments signal to Schwann cells to begin actin sphere formation and axon disintegration. If the process is disrupted, axon disintegration is slowed and axon fragments impair nerve regeneration.
A new study reports mice that lack the protein Alix develop hydrocephalus.
A new study reports a gene expressed in the amygdala plays a crucial role in coordinating axon growth and development.