Pupil size and salivary alpha-amylase could be biological indicators for autonomic dysfunction in children with autism spectrum disorder.
A new Caltech study suggests that specific changes in an overactive immune system can indeed contribute to autism-like behaviors in mice, and that in some cases, this activation can be related to what a developing fetus experiences in the womb.
A low dose of the sedative clonazepam alleviated autistic-like behavior in mice with a mutation that causes Dravet syndrome in humans.
Researchers studying interventions for adolescents and young adults with autism are reporting today that there is insufficient evidence to support findings, good or bad, for the therapies currently used.
Scientists have now identified a specific dysfunction in neuronal circuits that is caused by autism. The scientists also report about their success in reversing these neuronal changes in mouse models.
Neuroscientists take the first step toward deciphering the connection between general brain function and emergent behavioral patterns in autism. Study shows that autistic adults have unreliable neural sensory responses to visual, auditory and somatosensory, or touch, stimuli.
Some children who are accurately diagnosed in early childhood with autism lose the symptoms and the diagnosis as they grow older, a study supported by the National Institutes of Health has confirmed.
A study shows another family of proteins linked to neurodevelopmental disorders regulates the function of neuroligins and neurexins in order to suppress the development of inhibitory synapses.
Based on prior findings, researchers propose that depressed levels of insulin-like growth factor (IGF) in the blood of newborns could potentially serve as a biomarker for the later development of autism.