A new phenotype atlas assisted researchers in identifying 30 genes associated with an increased risk of developing schizophrenia. Single-cell RNA sequencing revealed an essential role for the transcription factor znf536 in the development of forebrain neurons associated with social behavior and stress.
Animal-assisted therapy improves social behaviors and reduces negative emotions in those with TBI. The findings report those with brain injuries exposed to AAT experienced higher social engagement, satisfaction and motivation following therapy sessions.
A newly developed augmented reality smartphone app may help reduce the fear of spiders for arachnophobes.
University of Basel researchers reveal the volume of the anterior insula, a brain area associated with emotion and empathy, is larger in boys who express higher levels of callous-unemotional traits.
Researchers have identified that some physical diseases tend to occur more frequently in young people who have previously suffered from certain mental disorders.
Researchers have designed two proteins that are able to stabilize cell scaffolding linking to muscle fibers, restoring muscle structure and function. The two proteins also were able to significantly prolong survival in animal models of muscular dystrophy.
A new study reports long distance neural connections can target pools of stem cells in their niche and stimulate them to produce subtypes of olfactory bulb neurons.
A new mathematical model has shown how different gene variants enable random diversity in neurons.
Using exercise programs that help boost motor skills can help reduce problems with impulse control and aid cognitive development in children born preterm.
University of Basel bioethicists have outlined a new biosecurity framework for neurotechnology. They call for regulations to protect the mental privacy and integrity of those the technologies are used on.
Probiotics can support the effects of antidepressants to help relieve symptoms of depression.
Researchers have uncovered a link between the Nlgn3 gene and the oxytocin signaling pathway in mouse models of ASD. Nlgn3 disrupts the oxytocin signaling pathway in neurons in the reward system, consequentially resulting in reduced social interactions in mice.