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TREM2

Image shows a jigsaw in the shape of a head.

Featured Neurology Neuroscience

·May 30, 2017·5 min read

Phagocytes in the Brain: Good or Bad?

Stimulating microglial cells could delay the onset of some neurodegenerative diseases, researchers report.

Featured Genetics Neurology Open Neuroscience Articles

·December 20, 2016·4 min read

The Molecular Roots of Alzheimer’s

Findings may help to shed light on how Alzheimer's and other neurodegenerative diseases develop.

Featured Neurology

·December 14, 2016·4 min read

Early Signs of Alzheimer’s Detected in Cerebrospinal Fluid

Researchers have discovered an early immune response in people with a genetic predisposition to Alzheimer's disease.

Featured Genetics Neurology Open Neuroscience Articles

·July 21, 2016·4 min read

Three Alzheimer’s Genetic Risk Factors Linked to Immune Cell Dysfunction

Researchers report the TREM2 mutation can derail the immune cell's plaque-clearing activity.

Image shows amyloid beta attacking a cell.

Featured Neurology

·June 15, 2016·8 min read

A New Role For Amyloid Beta in Alzheimer’s

Researchers have developed a new technique to measure levels of amyloid beta in the brain. They have also discovered new insights into why mutations of the TREM2 gene increases the risk of developing Alzheimer's disease.

Featured Neurology

·May 19, 2016·3 min read

Immune Cells May Protect Against Alzheimer’s

Researchers report microglia may actually protect the brain against Alzheimer's disease by containing the spread of amyloid plaques.

Featured Neurology Open Neuroscience Articles

·March 7, 2016·3 min read

Does A ‘Western Diet’ Increase Alzheimer’s Risk?

Having a sedentary lifestyle and eating a western diet is linked to an increased risk of developing Alzheimer's disease, a new study reports.

Diagram shows how TREM2 attaches to neurons.

Featured Neurology Open Neuroscience Articles

·March 5, 2016·5 min read

Early Biomarkers for Alzheimer’s Defined

Researchers have identified a biomarker that is associated with the activation of an innate immune response to neural damage during the early stages of Alzheimer's disease.