Researchers report snacking can raise inflammation and contribute to metabolic diseases.
UCSF researchers report the food and beverage industries push sugary products while obfuscating the significant health issues added sugars can cause. The findings shed light on sugars' link to disease and exposes industrial tactics to downplay the public health risks of diets too high in sugar.
Carbohydrate consumption from sugary drinks and snacks does not help to improve mood. The findings may help increase public awareness that the 'sugar rush' is a myth and help inform health policies to promote healthier alternatives.
A new brain training game in which players navigate a grocery store, earning rewards for selecting healthy food options, may help to reduce the desire to give into sugar cravings. Participants who had higher initial preferences for sugary foods lost as much as 3.1% body weight following daily gameplay.
Rats exposed to blue light at night were more prone to indulge in sugary snacks than regular foods.
Short term increases in sugar consumption raise the susceptibility to colitis and worsen symptoms of inflammatory bowel disease in mouse models.
From reducing memory capability to increasing the risk of diabetes and obesity, researchers investigate how sugar affects the brain and body.
If you are prone to depression, a new study suggests you should avoid indulging on dietary sugars. Added sugars have a pro-inflammatory effect on depressive disorders.
After 12 days of sugar intake, researchers noted major changes in both the dopamine and opioid systems of pigs' brains. Alterations in the opioid system were seen following the very first intake of sugar.
Taste perception appears to be controlled by dopamine in fruit fly models. Tracing the neural pathway, researchers found the same pathways were associated with controlling learning and memory. The network also appears to enhance taste sensations. Researchers also discovered eating lots of sugar suppresses sweet taste perception.
Sweet tasting foods don't only trigger the taste buds, they also switch on a neurological pathway that begins in the gut. In the intestines, signals of sugar ingestion travel to the brain, sparking an appetite for more sweet foods. However, this pathway only responds to sugars, not artificial sweeteners.
A mint product that contained gymnemic acids from the Gymnema sylvestre plant significantly reduced the intake of high-sugar sweet foods compared to a placebo. For those with a sweet tooth, the mint significantly decreased the pleasantness and desire for eating more sugar-rich foods. The product may be useful in helping people reduce sugar consumption.
