Technique could help those with C6, C7 spinal cord injuries. Surgeons at Washington University School of Medicine in St. Louis...
Researchers from the Huck Institutes’ Center for Cellular Dynamics, led by Center director Melissa Rolls, have found that a neuroprotective...
Researchers have identified an underlying cause of immune suppression in people with high level spinal cord injuries.
A training regimen to adjust the body's motor reflexes may help improve mobility for some people with incomplete spinal cord injuries. During training, the participants were instructed to suppress a knee jerk-like reflex elicited by a small shock to the leg. Those able to calm hyperactive reflexes saw improvements in their walking.
Researchers in France and Sweden have discovered how one of the body’s own proteins is involved in generating chronic pain...
Using electrochemical stimulation and robot assisted rehabilitation techniques, researchers restore walking ability in a paraplegic rat. The study reports reorganization of neural branching in the reticular formation leads to new connections and is key to motor skill recovery.
Study could help pave the way for new research for neurodegenerative conditions such as motor neuron disease and muscular dystrophy.
A new study reports on how signals from the spinal cord adjust the sensitivity of hair cells in the inner ear to accommodate shifts in head position and to insure balance is maintained.
Deleting the enzyme PTEN allowed neurology researchers to regenerate corticospinal tract neurons after spinal cord injuries in rodents.
The researchers claim Taxol was effective in promoting regeneration of injured spinal cord nerve cells in rats following spinal cord lesions. Only a few weeks following the spinal cord lesions and Taxol application, rats showed significant improvement in movements.
Injecting a patient's own bone marrow derived stem cells significantly improved motor function within weeks in those with spinal cord injuries.
Inhibitory neurons in the spinal cord lose their connections to motor neurons in mouse models of ALS. While no connection between this deterioration to the development of ALS has been made, researchers say the loss of inhibitory signals could explain why motor neurons die in those with ALS.
